Buy online CAS 94-63-3 Pralidoxime Iodide antibiotic

After organophosphate insecticides enter the body, they combine with cholinesterase in the body, form phosphoacylase and lose the hydrolysis of acetylcholine, so the accumulation of acetylcholine occurs in the body, and a series of poisoning symptoms appear. Phosphoryl iodide can combine with phosphoryl group of phosphorylated cholestase to dissociate cholinesterase and restore the ability of hydrolyzing acetylcholine. It can also directly combine with organophosphate ester to produce non-toxic phosphorylated cholestase iodide and discharge it out of the body. Phosphodesidine iodide only has an effect on phosphoacylcholestase that has recently formed, but after a few hours, phosphoacylcholinesterase has been "aging", the enzyme activity is difficult to recover, so the application of such drugs in the treatment of early organophosphorus poisoning effect is better, but the treatment of chronic poisoning is ineffective. Selective detoxification of organophosphorus poisoning: such as 1605, 1059, tepu, ethyl thiophos better curative effect; But to dichlorvos, dimethoate, trichlorfon, malathion effect is poor or ineffective; It has no effect on diazinon, meflonophos, propylamine flonophos and octadecphos poisoning. For mild organophosphorus poisoning, this product or atropine can be used alone to control symptoms; For moderate and severe poisoning, it has almost no effect on the accumulated acetylcholine in the body, so it must be combined with atropine. After intravenous administration, the effective concentration in the blood is soon reached. In large doses, it can also enter the brain tissue through the blood-cerebrospinal fluid barrier and be discharged quickly by the kidney without accumulation poisoning. Patients with a rapid injection of 2G May also have toxic reactions

Buy online CAS 94-63-3 Pralidoxime Iodide Antibiotic

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